Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV003112759 | SCV003789900 | pathogenic | not provided | 2023-11-25 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Val40*) in the GFM1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in GFM1 are known to be pathogenic (PMID: 16632485, 17160893). This variant is present in population databases (no rsID available, gnomAD 0.007%). This variant has not been reported in the literature in individuals affected with GFM1-related conditions. ClinVar contains an entry for this variant (Variation ID: 2421099). For these reasons, this variant has been classified as Pathogenic. |
Gene |
RCV003112759 | SCV004168239 | uncertain significance | not provided | 2023-04-19 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Has not been previously published as pathogenic or benign to our knowledge |
Baylor Genetics | RCV003459779 | SCV004199332 | likely pathogenic | Hepatoencephalopathy due to combined oxidative phosphorylation defect type 1 | 2023-11-23 | criteria provided, single submitter | clinical testing |