Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Blueprint Genetics | RCV001075118 | SCV001240729 | likely pathogenic | Retinal dystrophy | 2018-08-22 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001862596 | SCV002127754 | pathogenic | Familial aplasia of the vermis; Meckel-Gruber syndrome; Nephronophthisis | 2023-05-13 | criteria provided, single submitter | clinical testing | ClinVar contains an entry for this variant (Variation ID: 866808). For these reasons, this variant has been classified as Pathogenic. This variant has not been reported in the literature in individuals affected with CEP290-related conditions. This sequence change creates a premature translational stop signal (p.Asp450Argfs*3) in the CEP290 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in CEP290 are known to be pathogenic (PMID: 16909394, 17345604, 20690115). This variant is present in population databases (rs774490795, gnomAD 0.0009%). |
Baylor Genetics | RCV003469277 | SCV004216760 | likely pathogenic | Bardet-Biedl syndrome 14 | 2022-03-23 | criteria provided, single submitter | clinical testing | |
Gene |
RCV004726871 | SCV005331903 | likely pathogenic | not provided | 2023-10-18 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Has not been previously published as pathogenic or benign to our knowledge; This variant is associated with the following publications: (PMID: 31964843) |