Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001953901 | SCV002247000 | pathogenic | Familial aplasia of the vermis; Meckel-Gruber syndrome; Nephronophthisis | 2023-08-29 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Glu610Aspfs*7) in the CEP290 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in CEP290 are known to be pathogenic (PMID: 16909394, 17345604, 20690115). This variant is present in population databases (no rsID available, gnomAD 0.001%). This premature translational stop signal has been observed in individual(s) with clinical features of Leber congenital amaurosis (PMID: 21153841). ClinVar contains an entry for this variant (Variation ID: 1457703). For these reasons, this variant has been classified as Pathogenic. |
Fulgent Genetics, |
RCV002484803 | SCV002793601 | pathogenic | Leber congenital amaurosis 10; Meckel syndrome, type 4; Senior-Loken syndrome 6; Joubert syndrome 5; Bardet-Biedl syndrome 14 | 2022-01-05 | criteria provided, single submitter | clinical testing | |
Baylor Genetics | RCV003471185 | SCV004216655 | pathogenic | Bardet-Biedl syndrome 14 | 2024-02-22 | criteria provided, single submitter | clinical testing |