Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000529924 | SCV000634664 | likely benign | Familial aplasia of the vermis; Meckel-Gruber syndrome; Nephronophthisis | 2024-12-26 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV000765112 | SCV000896334 | uncertain significance | Leber congenital amaurosis 10; Meckel syndrome, type 4; Senior-Loken syndrome 6; Joubert syndrome 5; Bardet-Biedl syndrome 14 | 2022-03-15 | criteria provided, single submitter | clinical testing | |
Gene |
RCV002469187 | SCV002765810 | uncertain significance | not provided | 2022-06-14 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; Has not been previously published as pathogenic or benign to our knowledge |
Natera, |
RCV001272013 | SCV001453641 | uncertain significance | Leber congenital amaurosis | 2020-06-14 | no assertion criteria provided | clinical testing | |
Prevention |
RCV004732937 | SCV005347149 | uncertain significance | CEP290-related disorder | 2024-09-15 | no assertion criteria provided | clinical testing | The CEP290 c.6392A>G variant is predicted to result in the amino acid substitution p.Glu2131Gly. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.095% of alleles in individuals of African descent in gnomAD, which may be too common to be an undocumented pathogenic variant. Although we suspect that this variant may be benign, at this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |