Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Center for Medical Genetics Ghent, |
RCV002275380 | SCV002559861 | likely pathogenic | See cases | 2022-08-12 | criteria provided, single submitter | clinical testing | The variant is not present in population database gnomAD nor in the MDSGene mutation database (https://www.mdsgene.org/) (PM2). In silico predictions are indicative of a pathogenic effect on the protein (PP3). The variant co-segregated with the affected family members (PP1). The phenotype is specific (PP4). In this particular domain there are very few benign missense variants (PP2). |
Ce |
RCV002511148 | SCV002821619 | likely pathogenic | not provided | 2022-12-01 | criteria provided, single submitter | clinical testing | ANO3: PP1:Strong, PM2, PP4, PS4:Supporting |
Institute of Human Genetics Munich, |
RCV003336516 | SCV004045965 | likely pathogenic | Dystonia 24 | 2023-08-30 | criteria provided, single submitter | clinical testing | |
Gene |
RCV002511148 | SCV005081165 | uncertain significance | not provided | 2023-12-09 | criteria provided, single submitter | clinical testing | Not observed at significant frequency in large population cohorts (gnomAD); In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; This variant is associated with the following publications: (PMID: 36228993) |