Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001039626 | SCV001203163 | pathogenic | Cerebral cavernous malformation 2 | 2023-09-20 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 838138). This variant is also known as NM_001029835.2:c.902C>G (p.S301*). This premature translational stop signal has been observed in individual(s) with clinical features of cerebral cavernous malformations (PMID: 23595507, 24466005). This variant is not present in population databases (gnomAD no frequency). This sequence change creates a premature translational stop signal (p.Ser280*) in the CCM2 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in CCM2 are known to be pathogenic (PMID: 18300272, 24689081). |
Gene |
RCV002511020 | SCV002820506 | pathogenic | not provided | 2024-01-13 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Has not been previously published as pathogenic or benign to our knowledge |