Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Sema4, |
RCV002258436 | SCV002529276 | uncertain significance | Fanconi anemia | 2021-11-12 | criteria provided, single submitter | curation | |
Labcorp Genetics |
RCV002258436 | SCV003249431 | uncertain significance | Fanconi anemia | 2024-01-05 | criteria provided, single submitter | clinical testing | This sequence change replaces serine, which is neutral and polar, with asparagine, which is neutral and polar, at codon 580 of the SLX4 protein (p.Ser580Asn). The frequency data for this variant in the population databases is considered unreliable, as metrics indicate poor data quality at this position in the gnomAD database. This variant has not been reported in the literature in individuals affected with SLX4-related conditions. ClinVar contains an entry for this variant (Variation ID: 1692001). An algorithm developed to predict the effect of missense changes on protein structure and function (PolyPhen-2) suggests that this variant is likely to be tolerated. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Ambry Genetics | RCV003289485 | SCV003987500 | uncertain significance | Inborn genetic diseases | 2023-06-12 | criteria provided, single submitter | clinical testing | The c.1739G>A (p.S580N) alteration is located in exon 8 (coding exon 7) of the SLX4 gene. This alteration results from a G to A substitution at nucleotide position 1739, causing the serine (S) at amino acid position 580 to be replaced by an asparagine (N). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |