Total submissions: 4
| Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
|---|---|---|---|---|---|---|---|---|
| Labcorp Genetics |
RCV002231195 | SCV002508991 | pathogenic | Short-rib thoracic dysplasia 11 with or without polydactyly | 2024-12-31 | criteria provided, single submitter | clinical testing | This sequence change replaces arginine, which is basic and polar, with tryptophan, which is neutral and slightly polar, at codon 182 of the WDR34 protein (p.Arg182Trp). This variant is present in population databases (rs555811074, gnomAD 0.006%). This missense change has been observed in individuals with short-rib thoracic dysplasia (PMID: 29068549, 29241935, 36653407; internal data). ClinVar contains an entry for this variant (Variation ID: 446625). An algorithm developed to predict the effect of missense changes on protein structure and function (PolyPhen-2) suggests that this variant is likely to be disruptive. Experimental studies have shown that this missense change affects WDR34 function (PMID: 36268591). For these reasons, this variant has been classified as Pathogenic. |
| Institute for Clinical Genetics, |
RCV003321641 | SCV004026493 | likely pathogenic | not provided | 2021-12-14 | criteria provided, single submitter | clinical testing | PP3, PM1, PM2_SUP, PP1_MOD |
| Dan Cohn Lab, |
RCV000515869 | SCV000612031 | pathogenic | Jeune thoracic dystrophy | 2017-06-01 | no assertion criteria provided | research | |
| University of Washington Center for Mendelian Genomics, |
RCV000515869 | SCV001479908 | likely pathogenic | Jeune thoracic dystrophy | no assertion criteria provided | research |