Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000004900 | SCV002206063 | uncertain significance | Cranioectodermal dysplasia 1 | 2023-08-10 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Variants that disrupt the consensus splice site are a relatively common cause of aberrant splicing (PMID: 17576681, 9536098). Studies have shown that this variant results in skipping of exon 6 and introduces a premature termination codon (PMID: 20493458). The resulting mRNA is expected to undergo nonsense-mediated decay. ClinVar contains an entry for this variant (Variation ID: 4637). This variant has been observed in individual(s) with IFT122-related conditions (PMID: 20493458). This variant is present in population databases (rs376595844, gnomAD 0.004%). This sequence change falls in intron 6 of the IFT122 gene. It does not directly change the encoded amino acid sequence of the IFT122 protein. RNA analysis indicates that this variant induces altered splicing and may result in an absent or disrupted protein product. |
Fulgent Genetics, |
RCV000004900 | SCV002779400 | uncertain significance | Cranioectodermal dysplasia 1 | 2022-05-26 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000004900 | SCV000025076 | pathogenic | Cranioectodermal dysplasia 1 | 2010-06-11 | no assertion criteria provided | literature only | |
Gene |
RCV000004900 | SCV000087024 | not provided | Cranioectodermal dysplasia 1 | no assertion provided | literature only |