Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Genetic Services Laboratory, |
RCV000499928 | SCV000595859 | pathogenic | Combined oxidative phosphorylation defect type 15 | 2016-05-10 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV003558416 | SCV004297616 | pathogenic | not provided | 2023-09-22 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Glu74Lysfs*3) in the MTFMT gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in MTFMT are known to be pathogenic (PMID: 21907147, 24461907). This variant is present in population databases (rs777725264, gnomAD 0.004%). This premature translational stop signal has been observed in individual(s) with MTFMT-related conditions (PMID: 24461907, 25911677). ClinVar contains an entry for this variant (Variation ID: 435899). For these reasons, this variant has been classified as Pathogenic. |
Gene |
RCV003558416 | SCV005080460 | pathogenic | not provided | 2023-12-12 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 25911677, 24461907) |