Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV002281927 | SCV002571314 | uncertain significance | not provided | 2022-03-10 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; This variant is associated with the following publications: (PMID: 23173898, 24963352, 31527525) |
Payam Genetics Center, |
RCV000087055 | SCV003918902 | pathogenic | Autosomal recessive nonsyndromic hearing loss 22 | 2023-03-01 | criteria provided, single submitter | clinical testing | The OTOA c.1879C>T (p.Pro627Ser) is a missens mutation and results at the protein level is a disfunctional or truncated protein, predicted lead to disease.This variant is not present in Iranian population databases. This variant as Pathogenic according to the ACMG classification. |
Labcorp Genetics |
RCV002281927 | SCV004297686 | pathogenic | not provided | 2024-01-18 | criteria provided, single submitter | clinical testing | This sequence change replaces proline, which is neutral and non-polar, with serine, which is neutral and polar, at codon 627 of the OTOA protein (p.Pro627Ser). This variant is present in population databases (rs587777133, gnomAD 0.03%). This missense change has been observed in individuals with deafness (PMID: 23173898). It has also been observed to segregate with disease in related individuals. ClinVar contains an entry for this variant (Variation ID: 100655). An algorithm developed to predict the effect of missense changes on protein structure and function (PolyPhen-2) suggests that this variant is likely to be disruptive. For these reasons, this variant has been classified as Pathogenic. |
OMIM | RCV000087055 | SCV000119869 | pathogenic | Autosomal recessive nonsyndromic hearing loss 22 | 2013-09-01 | no assertion criteria provided | literature only |