Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000023642 | SCV000797798 | pathogenic | Ellis-van Creveld syndrome | 2018-02-16 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001852025 | SCV002164018 | pathogenic | Ellis-van Creveld syndrome; Curry-Hall syndrome | 2023-03-18 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 30665). This premature translational stop signal has been observed in individual(s) with Ellis-van Creveld syndrome (PMID: 21815252, 26064711, 26818569, 29456477). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. This variant is present in population databases (rs146538906, gnomAD 0.003%). This sequence change creates a premature translational stop signal (p.Arg885*) in the EVC2 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in EVC2 are known to be pathogenic (PMID: 17024374, 19810119, 19876929). |
OMIM | RCV000023642 | SCV000044933 | pathogenic | Ellis-van Creveld syndrome | 2011-09-01 | no assertion criteria provided | literature only |