Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001763269 | SCV001990662 | uncertain significance | not provided | 2019-04-02 | criteria provided, single submitter | clinical testing | Not observed at a significant frequency in large population cohorts (Lek et al., 2016); The majority of missense variants in this gene are considered pathogenic (Stenson et al., 2014); In silico analysis, which includes protein predictors and evolutionary conservation, supports a deleterious effect; Has not been previously published as pathogenic or benign to our knowledge |
Labcorp Genetics |
RCV002034490 | SCV002127169 | uncertain significance | Nemaline myopathy 8 | 2020-12-11 | criteria provided, single submitter | clinical testing | Algorithms developed to predict the effect of missense changes on protein structure and function (SIFT, PolyPhen-2, Align-GVGD) all suggest that this variant is likely to be disruptive, but these predictions have not been confirmed by published functional studies and their clinical significance is uncertain. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. This variant has not been reported in the literature in individuals with KLHL40-related conditions. This variant is present in population databases (rs374038735, ExAC 0.003%). This sequence change replaces aspartic acid with histidine at codon 144 of the KLHL40 protein (p.Asp144His). The aspartic acid residue is highly conserved and there is a moderate physicochemical difference between aspartic acid and histidine. |