Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000411609 | SCV000485350 | likely pathogenic | Cohen syndrome | 2016-01-14 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000411609 | SCV002211081 | pathogenic | Cohen syndrome | 2024-07-11 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Thr3780Hisfs*98) in the VPS13B gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in VPS13B are known to be pathogenic (PMID: 15141358, 16648375, 20461111). This variant is present in population databases (no rsID available, gnomAD 0.01%). This variant has not been reported in the literature in individuals affected with VPS13B-related conditions. ClinVar contains an entry for this variant (Variation ID: 370117). For these reasons, this variant has been classified as Pathogenic. |
Athena Diagnostics | RCV004999361 | SCV005621179 | pathogenic | not provided | 2023-09-26 | criteria provided, single submitter | clinical testing | This variant is expected to result in the loss of a functional protein. The frequency of this variant in the general population is consistent with pathogenicity. (http://gnomad.broadinstitute.org) This variant has been identified in at least one individual tested at Athena Diagnostics with clinical features associated with this gene. |