Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000552220 | SCV000630890 | pathogenic | Cohen syndrome | 2024-07-01 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Gly2706Alafs*29) in the VPS13B gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in VPS13B are known to be pathogenic (PMID: 15141358, 16648375, 20461111). This variant is not present in population databases (gnomAD no frequency). This variant has not been reported in the literature in individuals affected with VPS13B-related conditions. ClinVar contains an entry for this variant (Variation ID: 459250). For these reasons, this variant has been classified as Pathogenic. |
Greenwood Genetic Center Diagnostic Laboratories, |
RCV001805152 | SCV002051475 | likely pathogenic | not provided | 2020-11-16 | criteria provided, single submitter | clinical testing | PVS1, PM2 |
Gene |
RCV001805152 | SCV005688055 | likely pathogenic | not provided | 2024-07-29 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Has not been previously published as pathogenic or benign to our knowledge |
Counsyl | RCV000552220 | SCV001132508 | likely pathogenic | Cohen syndrome | 2015-04-08 | no assertion criteria provided | clinical testing |