Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
MGZ Medical Genetics Center | RCV002289055 | SCV002581447 | pathogenic | Developmental and epileptic encephalopathy, 7 | 2022-02-21 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV003588801 | SCV004297362 | pathogenic | Early infantile epileptic encephalopathy with suppression bursts | 2023-03-26 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt KCNQ2 protein function. ClinVar contains an entry for this variant (Variation ID: 1709240). This missense change has been observed in individual(s) with developmental and epileptic encephalopathy (PMID: 27779742, 29588952). In at least one individual the variant was observed to be de novo. This variant is not present in population databases (gnomAD no frequency). This sequence change replaces tyrosine, which is neutral and polar, with histidine, which is basic and polar, at codon 284 of the KCNQ2 protein (p.Tyr284His). |
Channelopathy- |
RCV003315373 | SCV004015092 | not provided | Complex neurodevelopmental disorder | no assertion provided | literature only |