Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000878106 | SCV001020956 | likely benign | Developmental and epileptic encephalopathy, 30 | 2024-10-24 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002390852 | SCV002670884 | likely benign | Inborn genetic diseases | 2018-07-19 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |
Center for Genomics, |
RCV000878106 | SCV003920475 | likely benign | Developmental and epileptic encephalopathy, 30 | 2022-09-01 | criteria provided, single submitter | clinical testing | This variant has not been reported in the literature but is present in the Genome Aggregation Database (Highest reported MAF 0.04% (48/111448) (https://gnomad.broadinstitute.org/variant/21-44839122-G-A?dataset=gnomad_r2_1). This variant is present in ClinVar (Variation ID:707210). Evolutionary conservation and computational predictive tools for this variant are limited or unavailable. This variant is an intronic variant with no predicted change in the amino acid sequence but may have an unknown effect on splicing. Splice prediction tools do not suggest that this variant may affect splicing. However, further studies are needed to understand its impact. In summary, data on this variant is insufficient for disease classification. Therefore, the clinical significance of this variant is uncertain. |