Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001552346 | SCV001773016 | likely pathogenic | not provided | 2021-02-04 | criteria provided, single submitter | clinical testing | In silico analysis, which includes protein predictors and evolutionary conservation, supports a deleterious effect; This variant is associated with the following publications: (PMID: 30011118, 27025581) |
Women's Health and Genetics/Laboratory Corporation of America, |
RCV003155281 | SCV003845119 | likely pathogenic | Lamellar ichthyosis | 2023-02-21 | criteria provided, single submitter | clinical testing | Variant summary: CYP4F22 c.727C>T (p.Arg243Cys) results in a non-conservative amino acid change in the encoded protein sequence. Five of five in-silico tools predict a damaging effect of the variant on protein function. The variant allele was found at a frequency of 2.4e-05 in 251400 control chromosomes (gnomAD). c.727C>T has been reported in the literature in bi-allelic individuals affected with Lamellar Ichthyosis (example: Pigg_2016). To our knowledge, no experimental evidence demonstrating an impact on protein function has been reported. A different variant affecting the same amino acid (p.R243H) is classified pathogenic by our lab and in ClinVar. Two clinical diagnostic laboratories have submitted clinical-significance assessments for this variant to ClinVar after 2014 and classified the variant as pathogenic/likely pathogenic. Based on the evidence outlined above, the variant was classified as likely pathogenic. |
Institute for Human Genetics, |
RCV000678426 | SCV000804500 | pathogenic | Autosomal recessive congenital ichthyosis 5 | 2018-04-23 | no assertion criteria provided | clinical testing |