Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000054548 | SCV001106672 | benign | Nephronophthisis 16 | 2023-06-22 | criteria provided, single submitter | clinical testing | |
Revvity Omics, |
RCV000054548 | SCV003818638 | uncertain significance | Nephronophthisis 16 | 2019-08-07 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000054548 | SCV000083026 | pathogenic | Nephronophthisis 16 | 2013-08-01 | no assertion criteria provided | literature only | |
Reproductive Health Research and Development, |
RCV000054548 | SCV001142399 | likely pathogenic | Nephronophthisis 16 | 2020-01-06 | no assertion criteria provided | curation | NM_173551.3:c.1322A>G in the ANKS6 gene has an allele frequency of 0.006 in South Asian subpopulation in the gnomAD database. The FKTN c.1322A>G (p.Gln441Arg) variant has been detected in a family with polycystic kidney disease in homozygous state (PMID: 23793029). Functional studies revealed that p.Gln441Arg was deleterious for protein function (PMID: 23793029). Pathogenic computational verdict because pathogenic predictions from DANN, EIGEN, FATHMM-MKL, M-CAP, MutationAssessor and MutationTaster. Taken together, we interprete this variant as Pathogenic/Likely pathogenic. ACMG/AMP Criteria applied: PM3; PS3; PP3. |