Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001036401 | SCV001199762 | pathogenic | Fetal akinesia deformation sequence 1; Congenital myasthenic syndrome 10 | 2024-01-16 | criteria provided, single submitter | clinical testing | This sequence change falls in intron 1 of the DOK7 gene. It does not directly change the encoded amino acid sequence of the DOK7 protein. The frequency data for this variant in the population databases is considered unreliable, as metrics indicate poor data quality at this position in the gnomAD database. This variant has been observed in individual(s) with congenital myasthenic syndrome (PMID: 18626973, 20012313, 20458068, 23219351). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. This variant is also known as c.54+25_39del15 or IVS1+14del15. ClinVar contains an entry for this variant (Variation ID: 835508). For these reasons, this variant has been classified as Pathogenic. |
Ce |
RCV001091297 | SCV001247239 | pathogenic | not provided | 2020-06-01 | criteria provided, single submitter | clinical testing | |
Gene |
RCV001091297 | SCV002044211 | pathogenic | not provided | 2022-01-11 | criteria provided, single submitter | clinical testing | Published cDNA analysis confirmed a damaging effect, as this intronic variant resulted in the partial retention of exon 1 and a frameshift, leading to a premature stop codon (Selcen et al., 2008); This variant is associated with the following publications: (PMID: 20012313, 23219351, 18626973, 20458068, 32403337, 27535533) |
Baylor Genetics | RCV003467708 | SCV004194035 | likely pathogenic | Fetal akinesia deformation sequence 3 | 2024-02-27 | criteria provided, single submitter | clinical testing |