Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Color Diagnostics, |
RCV001176335 | SCV001340278 | likely benign | Familial hypercholesterolemia | 2018-11-11 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002451357 | SCV002616213 | likely benign | Cardiovascular phenotype | 2021-05-15 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |
All of Us Research Program, |
RCV004006288 | SCV004836397 | likely benign | Hypercholesterolemia, autosomal dominant, 3 | 2023-06-26 | criteria provided, single submitter | clinical testing | |
GENin |
RCV001176335 | SCV005441505 | likely benign | Familial hypercholesterolemia | 2023-11-30 | criteria provided, single submitter | clinical testing | This is a synonymous (silent) variant that is not predicted by SpliceAI to impact splicing. In addition, it occurs at a nucleotide that is not conserved. Therefore this variant has been classified as Likely Benign (BP4, BP7). |
Prevention |
RCV004538401 | SCV004711904 | likely benign | PCSK9-related disorder | 2023-12-06 | no assertion criteria provided | clinical testing | This variant is classified as likely benign based on ACMG/AMP sequence variant interpretation guidelines (Richards et al. 2015 PMID: 25741868, with internal and published modifications). |