Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001479038 | SCV001683329 | likely benign | Hypercholesterolemia, autosomal dominant, 3 | 2024-08-08 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002396139 | SCV002698634 | likely benign | Cardiovascular phenotype | 2019-12-18 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |
Fulgent Genetics, |
RCV001479038 | SCV002799974 | likely benign | Hypercholesterolemia, autosomal dominant, 3 | 2021-10-11 | criteria provided, single submitter | clinical testing | |
All of Us Research Program, |
RCV001479038 | SCV004823450 | likely benign | Hypercholesterolemia, autosomal dominant, 3 | 2023-10-02 | criteria provided, single submitter | clinical testing | |
GENin |
RCV004820204 | SCV005441509 | likely benign | Familial hypercholesterolemia | 2023-05-29 | criteria provided, single submitter | clinical testing | This is a synonymous (silent) variant that is not predicted by SpliceAI to impact splicing. In addition, it occurs at a nucleotide that is not conserved. Therefore this variant has been classified as Likely Benign (BP4, BP7). |