Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001443835 | SCV001646820 | likely benign | Hypercholesterolemia, autosomal dominant, 3 | 2020-06-03 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002405040 | SCV002712556 | likely benign | Cardiovascular phenotype | 2021-08-07 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |
Color Diagnostics, |
RCV003581791 | SCV004358824 | likely benign | Familial hypercholesterolemia | 2021-08-23 | criteria provided, single submitter | clinical testing | |
All of Us Research Program, |
RCV001443835 | SCV004845964 | likely benign | Hypercholesterolemia, autosomal dominant, 3 | 2023-12-01 | criteria provided, single submitter | clinical testing | |
GENin |
RCV003581791 | SCV005441518 | likely benign | Familial hypercholesterolemia | 2023-06-01 | criteria provided, single submitter | clinical testing | This is a synonymous (silent) variant that is not predicted by SpliceAI to impact splicing. In addition, it occurs at a nucleotide that is not conserved. Therefore this variant has been classified as Likely Benign (BP4, BP7). |