Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000699023 | SCV000827717 | uncertain significance | Autosomal recessive ataxia, Beauce type; Emery-Dreifuss muscular dystrophy 4, autosomal dominant | 2022-03-18 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Algorithms developed to predict the effect of missense changes on protein structure and function output the following: SIFT: "Tolerated"; PolyPhen-2: "Benign"; Align-GVGD: "Class C0". The cysteine amino acid residue is found in multiple mammalian species, which suggests that this missense change does not adversely affect protein function. ClinVar contains an entry for this variant (Variation ID: 576512). This variant has not been reported in the literature in individuals affected with SYNE1-related conditions. This variant is present in population databases (rs757688642, gnomAD 0.05%). This sequence change replaces arginine, which is basic and polar, with cysteine, which is neutral and slightly polar, at codon 7086 of the SYNE1 protein (p.Arg7086Cys). |
Gene |
RCV001766519 | SCV001999790 | uncertain significance | not provided | 2019-12-05 | criteria provided, single submitter | clinical testing | In silico analysis, which includes protein predictors and evolutionary conservation, supports a deleterious effect; Has not been previously published as pathogenic or benign to our knowledge |
Revvity Omics, |
RCV001766519 | SCV003826465 | uncertain significance | not provided | 2021-11-26 | criteria provided, single submitter | clinical testing | |
Mayo Clinic Laboratories, |
RCV001766519 | SCV005408043 | uncertain significance | not provided | 2024-04-25 | criteria provided, single submitter | clinical testing | BP4 |