Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV003089066 | SCV002481237 | likely benign | Dyskeratosis congenita, autosomal dominant 2; Idiopathic Pulmonary Fibrosis | 2022-05-19 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004558901 | SCV002720468 | likely benign | Dyskeratosis congenita | 2020-09-14 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |
Center for Genomics, |
RCV003224621 | SCV003920546 | likely benign | Acute myeloid leukemia; Dyskeratosis congenita, autosomal dominant 2; Pulmonary fibrosis and/or bone marrow failure, Telomere-related, 1; Melanoma, cutaneous malignant, susceptibility to, 9 | 2022-10-12 | criteria provided, single submitter | clinical testing | This variant has not been reported in the literature and is not present in large control databases. This variant is present in ClinVar (Variation ID: 1670589). This is a silent variant and does not change the amino acid and is not predicted to impact splicing; additionally, this nucleotide position is not well-conserved evolutionarily, reducing the probability that this variant is disease-causing. In summary, data on this variant suggests that this variant does not cause disease but requires further evidence. Therefore, this variant is classified as likely benign. |