Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV003239070 | SCV003936391 | pathogenic | not provided | 2024-07-11 | criteria provided, single submitter | clinical testing | Identified in 2 siblings with generalized epilepsy who also harbored a second GABRA5 variant and in an individual with febrile and absence seizures (PMID: 30033060, 35627257); Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 35627257, 30033060) |
Labcorp Genetics |
RCV003779867 | SCV004573275 | pathogenic | Epilepsy, childhood absence 2; Febrile seizures, familial, 8 | 2023-11-06 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Phe151Serfs*19) in the GABRG2 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in GABRG2 are known to be pathogenic (PMID: 22539854, 22750526, 24407264). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with seizures (PMID: 35627257). ClinVar contains an entry for this variant (Variation ID: 2507179). For these reasons, this variant has been classified as Pathogenic. |