Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ocular Genomics Institute, |
RCV001376464 | SCV001573613 | uncertain significance | Retinitis pigmentosa 12 | 2021-04-08 | criteria provided, single submitter | research | The CRB1 c.1733T>A variant was identified in an individual with retinitis pigmentosa with a presumed recessive inheritance pattern. Through a review of available evidence we were able to apply the following criteria: PM3, PM2. Based on this evidence we have classified this variant as Variant of Uncertain Significance. |
Labcorp Genetics |
RCV002549196 | SCV003523993 | pathogenic | Retinitis pigmentosa 12; Leber congenital amaurosis 8 | 2024-01-16 | criteria provided, single submitter | clinical testing | This sequence change replaces valine, which is neutral and non-polar, with glutamic acid, which is acidic and polar, at codon 578 of the CRB1 protein (p.Val578Glu). This variant is present in population databases (no rsID available, gnomAD 0.0009%). This missense change has been observed in individuals with CRB1-related conditions (PMID: 15459956, 23449718, 33921607). ClinVar contains an entry for this variant (Variation ID: 812298). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt CRB1 protein function with a positive predictive value of 80%. For these reasons, this variant has been classified as Pathogenic. |
Sharon lab, |
RCV001002992 | SCV001161046 | likely pathogenic | Leber congenital amaurosis | 2019-06-23 | no assertion criteria provided | research |