Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001388311 | SCV001589244 | pathogenic | Retinitis pigmentosa 12; Leber congenital amaurosis 8 | 2020-07-02 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Loss-of-function variants in CRB1 are known to be pathogenic (PMID: 10508521, 22065545, 23379534, 25412400, 26957898, 28041643, 29391521). This variant has not been reported in the literature in individuals with CRB1-related conditions. This variant is present in population databases (rs751935649, ExAC 0.001%). This sequence change creates a premature translational stop signal (p.Ser586Leufs*7) in the CRB1 gene. It is expected to result in an absent or disrupted protein product. |
Fulgent Genetics, |
RCV002504649 | SCV002813840 | likely pathogenic | Pigmented paravenous retinochoroidal atrophy; Retinitis pigmentosa 12; Leber congenital amaurosis 8 | 2021-12-07 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV003450082 | SCV004179960 | likely pathogenic | Leber congenital amaurosis 8 | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV003450083 | SCV004179961 | likely pathogenic | Pigmented paravenous retinochoroidal atrophy | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV003450081 | SCV004179962 | likely pathogenic | Retinitis pigmentosa 12 | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Baylor Genetics | RCV003450082 | SCV004211108 | likely pathogenic | Leber congenital amaurosis 8 | 2023-10-23 | criteria provided, single submitter | clinical testing |