Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Blueprint Genetics | RCV001075721 | SCV001241349 | pathogenic | Retinal dystrophy | 2019-05-09 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001862628 | SCV002245520 | pathogenic | Retinitis pigmentosa 12; Leber congenital amaurosis 8 | 2022-09-27 | criteria provided, single submitter | clinical testing | This variant is not present in population databases (gnomAD no frequency). This sequence change creates a premature translational stop signal (p.Cys591Serfs*29) in the CRB1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in CRB1 are known to be pathogenic (PMID: 10508521, 22065545, 23379534, 25412400, 26957898, 28041643, 29391521). This premature translational stop signal has been observed in individual(s) with inherited retinal dystrophy (PMID: 23362850). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. It has also been observed to segregate with disease in related individuals. For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 867150). |
Genome- |
RCV003455418 | SCV004179966 | pathogenic | Leber congenital amaurosis 8 | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV003455419 | SCV004179967 | pathogenic | Pigmented paravenous retinochoroidal atrophy | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV003455417 | SCV004179968 | pathogenic | Retinitis pigmentosa 12 | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Faculty of Health Sciences, |
RCV001257862 | SCV001434709 | pathogenic | Autosomal recessive retinitis pigmentosa | 2013-01-30 | no assertion criteria provided | literature only |