Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Mendelics | RCV000986492 | SCV001135504 | pathogenic | Leber congenital amaurosis 1 | 2019-05-28 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001042752 | SCV001206453 | pathogenic | Retinitis pigmentosa 12; Leber congenital amaurosis 8 | 2023-08-10 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Gly845Serfs*9) in the CRB1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in CRB1 are known to be pathogenic (PMID: 10508521, 22065545, 23379534, 25412400, 26957898, 28041643, 29391521). This variant is present in population databases (rs745348555, gnomAD 0.0009%). This premature translational stop signal has been observed in individual(s) with CRB1-related conditions (Invitae). ClinVar contains an entry for this variant (Variation ID: 801599). For these reasons, this variant has been classified as Pathogenic. |
Ce |
RCV003222187 | SCV003916540 | pathogenic | not provided | 2023-05-01 | criteria provided, single submitter | clinical testing | CRB1: PVS1, PM2 |
Genome- |
RCV003455002 | SCV004180060 | pathogenic | Leber congenital amaurosis 8 | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV003455001 | SCV004180061 | pathogenic | Retinitis pigmentosa 12 | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Baylor Genetics | RCV003455002 | SCV004211158 | pathogenic | Leber congenital amaurosis 8 | 2023-08-14 | criteria provided, single submitter | clinical testing |