Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Genetic Services Laboratory, |
RCV000146038 | SCV000193193 | pathogenic | Bilateral frontoparietal polymicrogyria | 2013-05-16 | criteria provided, single submitter | clinical testing | |
Courtagen Diagnostics Laboratory, |
RCV000146038 | SCV000236528 | pathogenic | Bilateral frontoparietal polymicrogyria | 2015-01-05 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV000762976 | SCV000893420 | pathogenic | Bilateral frontoparietal polymicrogyria; Polymicrogyria, bilateral perisylvian, autosomal recessive | 2018-10-31 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000806491 | SCV000946495 | pathogenic | not provided | 2023-11-25 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg476*) in the ADGRG1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in ADGRG1 are known to be pathogenic (PMID: 15044805, 20929962). This variant is present in population databases (rs587783652, gnomAD 0.01%). This premature translational stop signal has been observed in individual(s) with clinical features of bilateral frontoparietal polymicrogyria and bilateral frontoparietal polymicrogyria (PMID: 25922261, 29707406). ClinVar contains an entry for this variant (Variation ID: 158618). For these reasons, this variant has been classified as Pathogenic. |
Revvity Omics, |
RCV000806491 | SCV003816641 | pathogenic | not provided | 2022-04-12 | criteria provided, single submitter | clinical testing | |
Natera, |
RCV000146038 | SCV001452611 | pathogenic | Bilateral frontoparietal polymicrogyria | 2020-09-16 | no assertion criteria provided | clinical testing |