Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001062730 | SCV001227547 | pathogenic | not provided | 2024-01-25 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Gln74Hisfs*28) in the CERKL gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in CERKL are known to be pathogenic (PMID: 14681825, 23591405, 24043777). This variant is present in population databases (no rsID available, gnomAD 0.001%). This variant has not been reported in the literature in individuals affected with CERKL-related conditions. ClinVar contains an entry for this variant (Variation ID: 857117). For these reasons, this variant has been classified as Pathogenic. |
Ocular Genomics Institute, |
RCV001376311 | SCV001573411 | likely pathogenic | Retinitis pigmentosa 26 | 2021-04-08 | criteria provided, single submitter | research | The CERKL c.222del variant was identified in an individual with retinitis pigmentosa with a presumed recessive inheritance pattern. Through a review of available evidence we were able to apply the following criteria: PVS1, PM2. Based on this evidence we have classified this variant as Likely Pathogenic. |
Baylor Genetics | RCV001376311 | SCV004216808 | pathogenic | Retinitis pigmentosa 26 | 2024-03-29 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV001376311 | SCV005655348 | likely pathogenic | Retinitis pigmentosa 26 | 2024-01-10 | criteria provided, single submitter | clinical testing |